What You Need to Know About Human Growth Hormone Levels

Human growth hormone (HGH) levels vary by age and sex.

HGH is released into the bloodstream by the pituitary gland. The primary function of HGH is tissue growth in children. In adults, HGH is responsible for cellular repair and regeneration. HGH also plays a role in many bodily functions and processes, primarily fat metabolism. Therefore, it plays a role in maintaining muscle tone, health, and fitness.

Your HGH levels vary by age. As you might imagine, HGH levels are highest in children.

The normal range for HGH level is typically:

As men age, there is a natural decline in HGH levels. HGH levels can and do fluctuate throughout the day. This is why doctors prefer to talk about normal HGH levels as a range, rather than a fixed ng/mL level.

Furthermore, because HGH levels in the blood vary at any given time, doctors prefer to establish the normal ranges of HGH by age based on IGF-1 levels. IGF-1 is another hormone. It is created by the liver and stimulates HGH production. IGF-1 levels are more constant than HGH levels and give a clear picture of ability to produce and release HGH.

The following chart (provided by LabCorp our testing facility) gives the normal IGF-1 for men by age.

AGE In Years

Male (ng/mL)

31 to 35

88246

36 to 40

83233

41 to 45

75216

46 to 50

67205

51 to 55

61200

56 to 60

54194

61 to 65

49188

66 to 70

47192

71 to 75

41179

76 to 80

37172

81 to 85

34165

86 to 90

32166

>90

Not established

Normal levels of human growth hormone (HGH) levels vary by age and sex.

Low HGH levels may indicate problems with your pituitary gland. Below normal HGH levels by age in men can result in a reduced sense of wellbeing, increased fat, increased risk of heart disease, and weakening muscles and bones.

While it is rarer than low HGH levels, some people do suffer from HGH levels that are too high. If your HGH levels are higher than normal, it can result in a rare condition known as acromegaly, in which patients have swelling of the hands and feet and altered facial features. Extremely high levels of HGH in children can cause gigantism, or growth to unusually tall stature. In almost all cases of acromegaly or gigantism, the above normal HGH levels are the result of a pituitary tumor.

HGH levels in women also vary by age just as they do in men. Again, we use IGF-1 levels to establish normal HGH levels in women by age. The following chart shows the normal IGF-1 levels in women by age.

AGE In Years

Female (ng/mL)

31 to 35

73243

36 to 40

69227

41 to 45

62204

46 to 50

57195

51 to 55

53190

56 to 60

46172

61 to 65

42169

66 to 70

38163

71 to 75

37165

76 to 80

35165

81 to 85

34172

86 to 90

34178

>90

Not established

HGH levels fluctuate throughout the day, so normal levels of HGH are expressed in a range, rather than a fixed level.

In women, HGH levels drop to their lowest after menopause. However, sometimes women who are still in their childbearing years, might require growth hormone therapy. What impact does growth hormone have on pregnancy? There have been some studies that indicate for women who are having trouble getting pregnant, growth hormone therapy may increase fertility. Once a woman becomes pregnant, her body automatically produces excessive HGH and IGF-1 for the developing fetus. Supplemental HGH is not recommended for pregnant women.

HGH levels decrease if you have a disease, injury, or genetic condition that impacts your pituitary glands ability to make HGH. HGH levels also decline naturally as men and women age. This natural drop in HGH as you grow older can result in age-related GHD.

Since your growth hormone levels can and do change throughout the day, instead of a test that measures the HGH level in your blood at any given time, we test for the level of IGF-1.

During the test a blood sample will be drawn from a vein in your arm. Since HGH levels fluctuate throughout the day, we do not test for HGH, but rather IGF-1. IGF-1 mirrors HGH excesses and deficiencies, but the level in the blood is stable throughout the day, making it a more useful indicator of average HGH levels than testing for HGH.

Before performing specific HGH testing, if the IGF-1 level is found to be normal for age and sex, growth hormone deficiency (GHD) can be ruled out, and more definitive testing is not necessary.

There could be some ways to naturally increase your HGH levels. Exercise, particularly strength training, or high intensity interval training can raise HGH levels. Here are some other ways that both men and women can naturally raise their HGH levels.

The above lifestyle changes could help keep your HGH level close to normal for your age and gender. However, if the results of your HGH or IGF-1 test indicate you have a growth hormone deficiency, or GHD, the only way that it can be treated is with hormone replacement therapy.

If your growth hormone level is determined to be below normal, depending on your age, gender and symptoms you will likely be prescribed growth hormone injections. Growth hormone injections are used to treat GHD in children and adults.

Growth hormone injections are only available with a doctors prescription. There are many anti-aging benefits of HGH replacement therapy. One of the main benefits of growth hormone replacement, is that it will improve your ability to burn fat and build lean muscle. This benefit alone will go a long way to making you look and feel younger.

However, the many benefits of HGH do not stop there. HGH therapy:

HGH therapy is a safe and effective way to treat lower than normal HGH levels.

Now that you know a bit more about normal and low HGH levels, why not contact us today, and find out more about the many life-changing benefits of HGH and other hormone replacement therapies.

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What You Need to Know About Human Growth Hormone Levels

Growth Hormone Therapy | Norditropin (somatropin) Injection

Selected Important Safety Information

Do not use Norditropin if: you have a critical illness caused by certain types of heart or stomach surgery, trauma or breathing (respiratory) problems; you are a child with Prader-Willi syndrome who is severely obese or has breathing problems including sleep apnea; you have cancer or other tumors; you are allergic to somatropin or any of the ingredients in Norditropin; your healthcare provider tells you that you have certain types of eye problems caused by diabetes (diabetic retinopathy); you are a child with closed bone growth plates (epiphyses).

Indications and Usage

What is Norditropin (somatropin) injection?

Norditropin is a prescription medicine that contains human growth hormone and is used to treat:

Important Safety Information (contd)

Before taking Norditropin, tell your healthcare provider about all of your medical conditions, including if you:

Tell your healthcare provider about all the medicines you take, including prescription and over-the-counter medicines, vitamins, and herbal supplements. Norditropin may affect how other medicines work, and other medicines may affect how Norditropin works.

How should I use Norditropin?

What are the possible side effects of Norditropin? Norditropin may cause serious side effects, including:

The most common side effects of Norditropin include:

Pleaseclick herefor NorditropinPrescribing Information.

Norditropin is a prescription medication.

Novo Nordisk provides patient assistance for those who qualify. Please call 1-866-310-7549 to learn more about Novo Nordisk assistance programs.

You are encouraged to report negative side effects of prescription drugs to the FDA. Visit http://www.fda.gov/medwatch, or call 1-800- FDA-1088.

Talk to your health care provider and find out if Norditropin is right for you or your child.

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Growth Hormone Therapy | Norditropin (somatropin) Injection

Igor Vovkovinskiy, Who At 7-Foot-8 Was The Tallest Man In The U.S., Has Died – NPR

Igor Vovkovinskiy, the country's tallest person at 7-foot-8, died in Minnesota at the age of 38. His family says he died of heart disease. Haraz N. Ghanbari/AP hide caption

Igor Vovkovinskiy, the country's tallest person at 7-foot-8, died in Minnesota at the age of 38. His family says he died of heart disease.

ROCHESTER, Minn. Igor Vovkovinskiy, the tallest man in the United States, has died. He was 38.

His family said the Ukrainian-born Vovkovinskiy died of heart disease on Friday at the Mayo Clinic in Rochester. His mother, Svetlana Vovkovinska, an ICU nurse at Mayo, initially posted about his death on Facebook.

Vovkovinskiy came to the Mayo Clinic in 1989 as a child seeking treatment. A tumor pressing against his pituitary gland caused it to secrete abnormal levels of growth hormone. He grew to become the tallest man in the U.S. at 7 feet, 8.33 inches (2 meters, 34.5 centimeters) and ended up staying in Rochester.

His older brother, Oleh Ladan of Brooklyn Park, told the Star Tribune of Minneapolis that Vovkovinskiy was a celebrity when he arrived from Ukraine because of his size and the flickering Cold War of the late 1980s. But Ladan said Vovkovinskiy "would have rather lived a normal life than be known."

Vovkovinskiy met President Barack Obama during a health care reform rally in 2009, when the president noticed him near the stage wearing a T-shirt that read, "World's Biggest Obama Supporter." Ben Garvin/AP hide caption

Vovkovinskiy appeared on "The Dr. Oz Show" and was called out by President Barack Obama during a campaign rally in 2009, when the president noticed him near the stage wearing a T-shirt that read, "World's Biggest Obama Supporter." In 2013, he carried the Ukrainian contestant onto the stage to perform in the Eurovision Song Contest.

When he was 27, Vovkovinskiy traveled to New York City and was declared America's tallest living person by a Guinness World Records adjudicator on Oz's show. He edged out a sheriff's deputy in Virginia by one-third of an inch.

He issued a plea in 2012 to cover the estimated $16,000 cost for specially made shoes that wouldn't cause him crippling pain. At the time, he said he hadn't owned a pair for years that fit his size 26, 10E feet. Thousands donated more than double what he needed. Reebok provided the custom shoes for free.

Vovkovinskiy was born Sept. 8, 1982, in Bar, Ukraine, to Vovkovinska and Oleksandr Ladan, according to Ranfranz and Vine Funeral Home, which is holding a memorial service on Saturday. His father died earlier.

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Igor Vovkovinskiy, Who At 7-Foot-8 Was The Tallest Man In The U.S., Has Died - NPR

Can what you eat save you from COVID-19? – The Jerusalem Post

Could good nutrition save people from developing severe COVID-19?

According to experts in the field, nutrition is the biggest coronavirus risk factor that not enough people are talking about.

We have two epidemics: obesity and COVID-19, said Dr. Mariela Glandt, a Harvard University and Columbia University trained endocrinologist and nutritionist who now lives in Israel and runs a clinic for diabetics in Ramat Aviv.

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She said, As long as the pandemic is still going on, anyone who cares about their health should do everything they can to improve the risk factors that they control among them diet.

A poor diet, like the modern American diet, with its junk food, ultra-processed starches and cheap fats, causes metabolic dysfunction that can be a disaster when its combined with the coronavirus, Glandt wrote in an eBook titled How to Eat in the Time of COVID-19 that she recently published with Ross Wollen and Jessica Apple.

The book was published by ASweetLife, which describes itself as the Internets trusted authority on the art of living well with diabetes.

Severe COVID-19 hospitalization, treatment in an intensive care unit, mechanical ventilation and even death has been associated with higher body mass index, the Centers for Disease Control has said.

Specifically, obesity defined by BMI increases the odds of hospitalization by 76%, Boaz showed in a paper that is soon to be published but has not yet been peer reviewed. She said the likelihood of ICU admission increases by 67%, mechanical ventilation by 119% and death by 37% all according to recent studies.

Moreover, a study that was published in the peer-reviewed journal PLOS One at the end of last month showed that people with high sugar values but who were not diagnosed with diabetes were also at risk of severe COVID-19 morbidity or mortality.

The researchers from Hebrew University of Jerusalem, Meuhedet Health Services, Jerusalem College of Technology and Hadassah-University Medical Center conducted a retroactive study among all individuals over the age of 18 who were insured by Meuhedet and contracted the virus between March and October 2020.

Of the 37,121 people who tested positive, 707 of them had severe disease, including 244 who died.

THE NORMAL blood sugar level in an adult is 70 to 100 milligrams per deciliter (mg/dL) of blood sugar after an eight-hour fast. Patients with 105-125 mg/dL were 1.5 times more likely to have severe COVID than patients with sugar lower than 105. Patients with between 125-140 mg/dL were twice as likely to develop complications.

The aim of the study was to identify risk factors for severe coronavirus illness that can be treated ahead of time, explained Dr. Michal Shauly-Aharonov of the Jerusalem College of Technology.

Obesity, high blood pressure, diabetes and many forms of cardiovascular disease are symptoms of an underlying medical condition called insulin resistance, Glandt wrote in her book.

Insulin is the hormone that allows your body to utilize the glucose in the food that you eat. Normally, this is a healthy and natural process insulin levels rise when eating, and subside to very low levels between meals, Glandt explained. Insulin is a storage and growth hormone and it is critical that there be a balance between high and low levels.

But when you eat a diet full of sweets and starches, your insulin production can grow out of control. Eventually, your cells become resistant to insulin, dulling its effect, which just causes your body to produce even more of it, as your body struggles to move glucose [sugar] out of your blood and into your cells, she said.

High sugar levels mean there is no more room to store sugar in a persons cells and so the sugar stays in the blood. If a person stops bringing sugar into their body, their sugar level will decline.

Through her own clinic, she has managed to get 97% of clients off insulin through diet management. The average client loses eight kg. (18 lbs.) in the first six months. Moreover, some 65% of clients normalize their blood sugar to the extent that they are no longer considered diabetic.

She advocates for the ketogenic diet, which includes avoiding all seed oils, such as canola, soybean, sunflower and corn; avoiding all sugars; and keeping complex carbohydrates to a minimum.

Boaz, on the other hand, has said that a modified Mediterranean diet to achieve the right balance, which includes fish, nuts, hummus, tahini and refried beans, is high in fruits and vegetables, whole grains and olive oils and low in processed foods.

This particular pandemic highlighted the impact that obesity can have on the immune system, Boaz said.

She stressed that public health policy should be directed at improving diet quality, especially among youths who are much more likely to follow healthful practices if taught from an early age.

Logic would tell you, Boaz said, being adequately nourished and maintaining a healthy diet is always to your benefit.

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Can what you eat save you from COVID-19? - The Jerusalem Post

NZME Limited (NZX: NZM) Half Year Results to 30 June 2021 – sharechat.co.nz – sharechat

NZME Limited has today announced its financial results for the half year ended 30 June 2021, reporting Statutory Net Profit After Tax (NPAT) of $5.6 million, up 85% on the corresponding period in 2020.

NZME also announced growth in Operating Earnings before Interest, Tax, Depreciation and Amortisation (EBITDA) to $30.1 million for the half year, representing 4% growth in Operating EBITDA against the first half of 2020. Operating revenue was $172.5m which was 9% higher than the first half of 2020.

NZME is delighted to share a set of results that feature earnings growth and a further reduction in net debt, said NZME CEO Michael Boggs.

Emerging from the significant disruption encountered in 2020 NZME has maintained a steadfast focus on key strategic priorities. This has meant that as New Zealands many commercial sectors are steadily rebuilding their investment in audience engagement, NZMEs advertising revenues continue to approach the levels achieved in 2019, before the pandemic struck, said Boggs.

NZMEs Net Debt was reduced by a further $15.2 million during the half year to $18.6 million and is now below NZMEs target leverage ratio.

A rigorous commercial discipline and a continual focus on managing the cost base as business activity recovers has improved NZMEs ongoing Capital Management performance and has supported the continued strengthening of NZMEs Balance Sheet, said NZME Chairman Barbara Chapman.

NZME today announced that given the significant reduction in debt and based on the business outlook and NZMEs capital requirements the NZME board has declared a fully imputed and fully franked dividend of 3.0 cps.

Please see the links below for details

NZME 2021 Half Year Results - Cover Note

NZME 2021 Half Year Results NZX Form

NZME 2021 Half Year Results Announcement

NZME 2021 Half Year Results Presentation

NZME 2021 Consolidated Interim Financial Statements

Distribution Notice - NZX Form

Source: NZME Limited

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Ascendis Pharma A/S Announces Second Quarter 2021 Financial Results and Business Update Conference Call on August 25 – Stockhouse

COPENHAGEN, Denmark, Aug. 17, 2021 (GLOBE NEWSWIRE) -- Ascendis Pharma A/S (Nasdaq: ASND), a biopharmaceutical company that utilizes its innovative TransCon technologies to create product candidates that address unmet medical needs, today announced that the company will hold a conference call and live webcast on Wednesday, August 25, 2021 at 4:30 p.m. Eastern Time (ET) to review its second quarter 2021 financial results and provide a business update.

Conference Call Details

A live webcast of the conference call will be available on the Investors and News section of the Ascendis Pharma website at http://www.ascendispharma.com. A webcast replay will be available on this website shortly after conclusion of the event for 30 days.

About Ascendis Pharma A/S

Ascendis Pharma is applying its innovative platform technology to build a leading, fully integrated biopharma company focused on making a meaningful difference in patients’ lives. Guided by its core values of patients, science and passion, the company utilizes its TransCon technologies to create new and potentially best-in-class therapies.

Ascendis Pharma currently has a pipeline of three independent endocrinology rare disease product candidates and one oncology product candidate in clinical development. The company continues to expand into additional therapeutic areas to address unmet patient needs.

Ascendis is headquartered in Copenhagen, Denmark, with additional facilities in Heidelberg and Berlin, Germany, in Palo Alto and Redwood City, California, and in Princeton, New Jersey.

Please visit http://www.ascendispharma.com (for global information) or http://www.ascendispharma.us (for U.S. information).

Forward-Looking Statements

This press release contains forward-looking statements that involve substantial risks and uncertainties. All statements, other than statements of historical facts, included in this press release regarding Ascendis’ future operations, plans and objectives of management are forward-looking statements. Examples of such statements include, but are not limited to, statements relating to (i) Ascendis’ ability to apply its platform technology to build a leading, fully integrated biopharma company, (ii) Ascendis’ product pipeline and expansion into additional therapeutic areas and (iii) Ascendis’ expectations regarding its ability to utilize its TransCon technologies to create new and potentially best-in-class therapies. Ascendis may not actually achieve the plans, carry out the intentions or meet the expectations or projections disclosed in the forward-looking statements and you should not place undue reliance on these forward-looking statements. Actual results or events could differ materially from the plans, intentions, expectations and projections disclosed in the forward-looking statements. Various important factors could cause actual results or events to differ materially from the forward-looking statements that Ascendis makes, including the following: unforeseen safety or efficacy results in its oncology programs, TransCon hGH, TransCon PTH and TransCon CNP or other development programs; unforeseen expenses related to the development and potential commercialization of its oncology programs, TransCon hGH, TransCon PTH and TransCon CNP or other development programs, selling, general and administrative expenses, other research and development expenses and Ascendis’ business generally; delays in the development of its oncology programs, TransCon hGH, TransCon PTH and TransCon CNP or other development programs related to manufacturing, regulatory requirements, speed of patient recruitment or other unforeseen delays; dependence on third party manufacturers to supply study drug for planned clinical studies; Ascendis’ ability to obtain additional funding, if needed, to support its business activities and the effects on its business from the worldwide COVID-19 pandemic. For a further description of the risks and uncertainties that could cause actual results to differ from those expressed in these forward-looking statements, as well as risks relating to Ascendis’ business in general, see Ascendis’ Annual Report on Form 20-F filed with the U.S. Securities and Exchange Commission (SEC) on March 10, 2021 and Ascendis’ other future reports filed with, or submitted to, the SEC. Forward-looking statements do not reflect the potential impact of any future in-licensing, collaborations, acquisitions, mergers, dispositions, joint ventures, or investments that Ascendis may enter into or make. Ascendis does not assume any obligation to update any forward-looking statements, except as required by law.

Ascendis, Ascendis Pharma, the Ascendis Pharma logo, the company logo and TransCon are trademarks owned by the Ascendis Pharma Group. August 2021 Ascendis Pharma A/S.

Investor contacts: Tim Lee Ascendis Pharma (650) 374-6343 tle@ascendispharma.com

Patti Bank Westwicke Partners (415) 513-1284 patti.bank@westwicke.com ir@ascendispharma.com

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Ascendis Pharma A/S Announces Second Quarter 2021 Financial Results and Business Update Conference Call on August 25 - Stockhouse

Study: Biosimilar rhGH Well Tolerated and Effective in Turner Syndrome – The Center for Biosimilars

A multicenter observational study of 348 children with Turner syndrome (TS) found that biosimilar recombinant human growth hormone (rhGH) therapy was safe and effective in real-life clinical practice.

The investigators reported on a subgroup of patients with TS from the PAtients TReated with Omnitrope (PATRO) Children study, a long-term, postmarketing surveillance study of children treated with biosimilar rhGH (Omnitrope; Sandoz).

TS is characterized by partial or complete loss of the second sex chromosome in females and is associated with comorbidities, including growth failure. TS has been treated with reference rhGH (Genotropin; Pfizer) since its approval in 1995. The biosimilar Omnitrope became available after approval by the European Medicines Agency and the FDA in 2006.

The investigators enrolled 348 patients being treated for TS at 130 centers across Europe. Their mean age was 9.0 (range, 0.7-18.5) years at baseline. Most (81.6%) were rhGH nave, and 90.2% were prepubertal. The mean (SD) treatment duration in the study was 38.5 (26.8) months.

No Unexpected Adverse Events

Safety was the primary end point of the study. The authors pointed out that rhGH is generally well tolerated in patients with TS; however, observational studies have reported greater risk for some adverse events (AEs) in patients with TS receiving rhGH. The AEs include intracranial hypertension, slipped capital femoral epiphyses, pancreatitis, and development or progression of scoliosis. The investigators wrote that their findings do not support an increased risk of these events due to rhGH therapy. However, noting the relatively short treatment duration in their study, they cautioned longer-term follow-up would be required to verify these safety findings.

Overall, approximately half of the patients (170; 48.9%) experienced AEs, most of which were mild or moderate. Twenty-five patients experienced treatment-related AEs, including headache, impaired glucose tolerance, increased insulin-like growth factor levels, and increased weight. One patient experienced scoliosis that was deemed treatment related.

Approximately half (172; 49.4%) of the patients discontinued the study. The most common reason (52 patients) for discontinuation was the attainment of adult height or bone age maturation. Six patients discontinued the study because of a lack of treatment response. Ten patients discontinued due to AEs, one of whom experienced a serious AE (intracranial hypertension) that was possibly related to treatment. The authors wrote that their safety findings show no evidence of an increased risk of developing unexpected AEs or new malignancies during rhGH treatment.

Substantial Height Gains

The investigators included 163 patients in their 3-year effectiveness cohort, 131 of whom were rhGH nave prior to the study. Most patients (98.5% of the rhGH-nave group and 90.6% of pretreated patients) were prepubertal.

Effectiveness was assessed by height velocity (HV), height standard deviation score (HSDS), and height velocity standard deviation score (HVSDS). The mean (SD) HV at 3 years was 5.2 (1.3) cm/year in prepubertal treatment-nave patients and 4.7 (1.4) cm/year in prepubertal pretreated patients. From baseline to 3 years, gains in HSDS were 1.17 in prepubertal treatment-nave patients and 0.04 in prepubertal pretreated patients. Mean increase in peak-centered HVSDS from baseline to 3 years was 3.94 in prepubertal rhGH-nave patients and 0.47 in prepubertal pretreated patients.

Adult height was reached by 51 patients (31.1%), 35 of whom were rhGH nave at baseline. In those 35 patients, the mean HSDS was 2.97 (1.03) at baseline, and at 3 years, the mean AHSDS was 2.02 (0.9). The AHSDS was similar in pretreated patients, at 2.10 (1.1).

These effectiveness findings are broadly in line with those from other observational studies in TS patients, according to the authors.

The authors concluded that treatment with biosimilar rhGH was well tolerated in patients with TS, and no unexpected safety signals were identified.

Several well-known AEs were observed, confirming the need for ongoing patient monitoring, they wrote. Although safety was their primary end point, they wrote that their effectiveness data showed substantial height gains were observed in prepubertal individuals over 3 years of treatment.

Reference

Backeljauw P, Kanumakala S, Loche S, et al. Safety and effectiveness of recombinant human growth hormone in children with Turner syndrome: data from the PATRO children study. Horm Res Paediatr. 2021;94(3-4):133-143. doi:10.1159/000515875

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Study: Biosimilar rhGH Well Tolerated and Effective in Turner Syndrome - The Center for Biosimilars

The prevalence of inorganic mercury in human cells increases during aging but decreases in the very old | Scientific Reports – Nature.com

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The prevalence of inorganic mercury in human cells increases during aging but decreases in the very old | Scientific Reports - Nature.com

Prognostic factors in patients with HR+/HER2 breast cancer | CMAR – Dove Medical Press

Introduction

Advances in screening and treatment paradigms for breast cancer has led to an overall decline in mortality rate in the past decade.1 The survival rate depends on stage of breast cancer at diagnosis, among other factors.2 The five-year survival rate for patients diagnosed with Stage IV breast cancer is 22%, for Stage III is 72% and Stage II is >90%.3 Clinical decision-making in breast cancer management relies on determination of receptor status, as therapies have been developed that specifically benefit patients depending on hormone receptor (HR) and human epidermal growth factor (HER2) receptor status.46 HR+/HER2 status is the most common molecular subtype, accounting for two-thirds of US female breast cancer cases.79

In addition to advancements in treatment options over time, prognosis of breast cancer is influenced by factors that indicate growth, invasion, and metastatic potential of disease, thereby informing disease course and clinical outcome.4 The HR+/HER2 subtype has been associated with improved survival compared with other subtypes in the metastatic setting, also indicating some prognostic relationship between survival and receptor status.4,10 Amongst HR+/HER2 subtype, survival is influenced by other disease-related factors such as tumor grade, site of the metastasis (eg bone, liver, lung, or brain), prior therapy, as well as patient-related factors (eg age, race).11,12

Although several studies have identified prognostic factors associated with survival, especially in the early breast cancer setting,1315 it remains unclear to what extent these factors impact prognosis in advanced breast cancer. Currently, there is no comprehensive summary assessing the collective available evidence and the strength of evidence for these prognostic factors among patients with HR+/HER2 advanced breast cancer that can aid clinical decision-making. Therefore, we conducted a systematic literature review (SLR) based on a pre-specified protocol to identify the prognostic factors associated with survival endpoints in patients with HR+/HER2 advanced breast cancer and qualitatively assess the evidence and its strength and consistency.

A SLR was conducted and reported in accordance with guidelines established by the Centre for Reviews and Dissemination (CRD),16 Preferred Reporting Items for Systematic Reviews and Meta-Analyses (PRISMA) statement,17 and Cochrane guidebook.18 Comprehensive searches were conducted in major electronic databases (MEDLINE, EMBASE, and Cochrane Controlled Register of Trials) to identify primary research studies published between January 1, 2010 and November 15, 2018. These were supplemented by searches of relevant conference proceedings (American Society of Clinical Oncology, European Society for Medical Oncology, European Cancer Organization, European Cancer Summit, Improving Care and Knowledge through Translational Research Breast Cancer Conference, The International Consensus Conference for Advanced Breast Cancer, San Antonio Breast Cancer Symposium, and American Association for Cancer Research) held in the two prior years to identify abstracts of interest. The primary publications related to the conference abstracts were searched. Relevant SLRs published recently were cross-checked to find additional studies. The search strategy was designed to include an extensive list of search terms (including MeSH/Emtree terms and natural language terms) which were broadly grouped into: 1) HR+/HER2- breast cancer, 2) advanced disease stage, 3) prognostic factors, 4) outcomesincluding tumor response, also referred to as objective response or clinical benefit, progression-free survival (PFS), overall survival (OS), and breast cancer-specific survival (BCSS). Disease terms included a combination of terms to identify advanced stage breast cancer in combination with terms specific to HR+/HER2- status.

Patients with HR+/HER2- advanced breast cancer were the population of interest for this SLR. However, there were limited studies that included this patient population exclusively. Besides, the proportion of patients with HR+/HER2- subtype widely varied across studies. Hence we decided to exclude studies where <50% of patients were either HR+ or HER2. Since the proportion of patients with advanced/metastatic breast cancer also varied across studies, we included studies where 80% of patients were diagnosed with advanced breast cancer. These eligibility criteria allowed for inclusion of studies with the population of interest, thus striking a balance between validity and generalizability of the review. Observational studies with sample size of 300 patients and RCTs with sample size of 300 patients were eligible for inclusion. Editorials, letters, commentaries, reviews, invitro-studies, and non-English publications were excluded. Since prognostic and predictive terms are used, sometimes incorrectly as interchangeable in literature,19 we excluded studies that reported the interaction p-value between a factor and treatment indicative of predictive association.

After removing duplicates, two reviewers independently screened abstracts and full-texts for eligibility. Disagreements were resolved by consensus or by a third reviewer. A single reviewer extracted all data, and a separate reviewer independently validated extracted data.

Strength of evidence was determined in terms of consistency of evidence, directionality of association, use of multivariable analyses, and strength of association based on effect size. If >50% of studies that assessed an association found it to be significant, then evidence was considered consistent. Similarly, if the direction of association was the same in >50% of studies that demonstrated a significant association, then directionality of association was deemed consistent. For example, negative progesterone receptor status was associated with worse survival in 100% of studies that reported a significant relationship. Based on hazard ratios (HR) calculated in univariate and multivariate analyses, the strength of associations was categorized as strong (HR3), moderate (HR=1.52.9), or weak (HR<1.5).20

Prognostic factors satisfying all the following criteria were deemed to have the strongest evidence of association with OS or PFS: i) consistency of evidence; ii) consistency in the direction of association; iii) at least >5 studies demonstrating a significant association. For example, circulating tumor cell (CTC) count showed the strongest evidence of association with OS in nine out of 10 studies (ie, achieved consistency based on >50% studies with a significant association) and showed consistency in direction of association as well as strength of association based on effect size. The Quality In Prognosis Studies (QUIPS) risk of bias assessment tool was used to assess study quality.21 Based on our understanding of the literature base and variability expected in the patient population and study design, we did not plan to conduct a meta-analysis of the relationship between prognostic factors and survival endpoints.

The PRISMA flow diagram summarizes the review (Figure 1). Overall, the SLR included 72 full-text articles and seven conference abstracts (Table 1).10,2298 The studies identified included retrospective data analyses (71%), prospective cohort studies (16.5%), studies with both retrospective and prospective data collections (2.5%), randomized controlled trials (RCTs) or clinical trials (7.5%), and post-hoc analyses of RCTs (2.5%). OS was the most commonly assessed endpoint (n=67), followed by PFS (n=33), while BCSS (n=5) and tumor response (n=3) were assessed less frequently. The majority of studies were conducted in Europe (n=38), followed by North America (n=15), Asia (n=18), Northern Africa (n=1), the Middle East (n=1), and five studies were multinational. One study did not report study location.

Figure 1 Preferred Reporting Items for Systematic Reviews and Meta-Analyses (PRISMA) flow diagram for study selection.

The median age of patients in the included studies ranged between 4468 years; age was not reported in 12 studies.28,31,55,66,8487,93,97 In 22 studies, the entire study population was HR+ and HER2, while in eight studies the proportion of patients with HR+ and/or HER2 status was between 8099%, and the remaining 49 studies included patients with HR+ and/or HER2 status ranging between 5079%.

Patients with breast cancer positive for progesterone, estrogen, or both receptors were deemed HR positive. The relationship between PR status and OS (n=10), PFS (n=2), and tumor response (n=1) was evaluated, with a significant association reported in 80% (n=8), 50% (n=1), and 100% (n=1) of studies, respectively.26,38,45,56,66,72,73,92,98 The association of PR status with BCSS was assessed in one study, and it did not report any significant relationship.40 Patients with negative PR status compared with positive were moderately associated with worse OS. The evidence was insufficient to assess the strength of association between PFS/tumor response and PR status.

The type of tumor grading system used was reported in only four studies that assessed OS. Two studies used the Scarff Bloom Richardson grading,52,88 one utilized the modied BloomRichardson grading,96 and the other study employed the Elston-Ellis modification of Scarff-Bloom-Richardson grading system.82

The relationship between tumor grade and OS (n=21), PFS (n=4), BCSS (n=3), and tumor response (n=1) was evaluated, with a significant association reported in 62% (n=13), 75% (n=3), 100% (n=1), and 100% (n=1) of studies, respectively.26,28,29,38,42,55,66,73,78,81,82,85,88,90 Survival was worse in patients with poorly to moderately differentiated tumors compared with well-differentiated tumors. Consistency in evidence and directionality of association was observed for all survival endpoints. Overall, the effect size of the association between tumor grade and survival endpoints was moderate.

Relationship between tumor size and OS (n=12) and BCSS (n=2) was evaluated, with a significant association reported in 42% (n=5) and 50% (n=1) of studies, respectively.28,38,81,90,92 No included study assessed the association between tumor size and PFS or tumor response. In four studies, large tumors (>5 cm diameter) were associated with worse survival,28,38,90,92 while one study showed improved OS in patients with T2 tumors (>2 cm and <5 cm) compared with T1 tumors (2 cm).81 Less than 50% of studies that assessed the association between tumor size and OS reported a significant association, although among those, directionality of evidence was consistent in the five studies. Overall, the effect size of the association between tumor size and survival endpoints ranged from weak-to-moderate.

The relationship between lymph node involvement and OS (n=11), PFS (n=1), and BCSS (n=2) was evaluated, with a significant association reported in 36% (n=4), 100% (n=1), and 100% (n=2) of studies, respectively.28,38,40,66,70,90

In two of four studies demonstrating a relationship with OS, N1, N2, and N3 categories were associated with better OS than patients with no lymph node involvement (N0);28,90 these studies involved stage IV de novo metastatic patients from the Surveillance, Epidemiology, and End Results (SEER) registry. The trend, however, was converse in the other two studies, among patients with metastatic disease with no prior diagnosis and another with recurrent disease after breast surgery or neoadjuvant chemotherapy, in which greater lymph node involvement was associated with greater risk of death.38,66 The two studies focusing on BCSS and the one study70 focusing on PFS also reported higher lymph node involvement was associated with greater risk of death.28,90 In summary, the directionality of association was inconsistent across studies assessing OS and lymph node involvement. Overall, the effect size of the association between lymph node involvement and survival endpoints was moderate.

In Gampenrieder et al,42 patients with lobular carcinoma (HR=3.44; 95% CI=1.0711.11; P=0.039) or other type of carcinoma (HR=3.19; 95% CI=1.059.70; P=0.041) were associated with 3-fold greater risk of death compared with ductal carcinoma; similar results were observed for PFS. The effect size of the association between histological type (lobular vs ductal) and survival endpoints was strong. The evidence of association was insufficient as a significant association was reported in only one of five studies with OS and two studies with PFS.

Relationship between CTC count and OS (n=10), PFS (n=10), and BCSS (n=1) was evaluated, with a significant association reported in 90% (n=9), 80% (n=8), and 0% (n=0) of studies, respectively.24,31,32,36,47,51,7476,83 The presence of a higher CTC count (5/7.5 mL whole blood) was consistently associated with poor OS and PFS.

The relationship between Ki67 expression and OS (n=7), PFS (n=4), and tumor response (n=1) was evaluated, with a significant association reported in 86% (n=6), 100% (n=4), and 100% (n=1) of studies, respectively.27,30,45,57,60,66,67,80 Studies did not consistently report the source of the Ki67 (primary or metastatic tumor site). High Ki67 expression was associated with worse OS, PFS, and tumor response. The thresholds for the Ki67 was inconsistent across studies, with a Ki67 index of 14% vs >14% being the most common.

The association of both CTCs and Ki67 with OS and PFS was harmonious with respect to consistency of evidence and directionality of association. Overall, the effect size of the association between these biomarkers and survival endpoints were moderate.

The relationship between de novo mBC and OS (n=5), PFS (n=3), and BCSS (n=1) was evaluated, with a significant association reported in 100% (n=5), 67% (n=2), and 0% (n=0) of studies, respectively.30,33,39,57,62,91 Four studies demonstrated longer OS in patients with mBC at diagnosis compared with recurrent breast cancer;30,39,57,91 while one study reported shorter OS in patients with de novo mBC.62 Similarly, one study showed longer PFS associated with patients with de novo mBC,30 while another study showed a reverse relationship.33

The association of de novo mBC with OS and PFS was consistent with respect to evidence. The directionality of association was consistent with OS but not with PFS. The effect size of the association between de novo mBC and survival endpoints ranged between weak to moderate.

The relationship between number of metastatic sites and OS (n=27), PFS (n=11), and BCSS (n=1) was evaluated, with a significant association reported in 89% (n=24), 55% (n=6), and 100% (n=1) of studies, respectively.10,2830,33,38,39,41,43,44,48,5155,57,59,60,66,69,71,75,79,80,89 Multiple metastases were associated with significantly worse OS and PFS. There were variations in the way comparisons between the number of metastatic sites were made across studies (eg, 1 vs >1; 3 vs >3); however, the multiple vs single site of metastases (ie, >1 vs 1) comparison was the most common. Most studies compared either the number of metastatic sites (eg, >1 vs 1) or types of sites/location of metastasis (eg, lungs vs brain, visceral vs non-visceral) However, three studies10,28,54 compared multiple metastatic sites (visceral, brain, skin, lymph nodes) to bone metastasis and found significantly greater risk of death associated with the former. Consistency in evidence and directionality of association was observed for OS and PFS. The effect size of the association between number of metastatic sites and survival endpoints ranged from moderate to strong, depending on the comparison groups.

Twenty-two of the 34 studies found a significant association between sites of metastasis and OS.10,29,30,32,36,38,39,4244,48,52,54,59,63,69,71,75,7880,89 Sites of metastasis were compared heterogeneously (eg, visceral vs non-visceral, visceral vs bone, hepatic vs no hepatic, brain vs no brain). Liver involvement was the most widely studied (n=1230,32,38,39,43,48,59,63,71,78,79,89), followed by brain/CNS (n=1410,29,30,38,43,48,54,61,63,69,7880,89), visceral (n=1310,30,36,4244,52,54,63,69,75,78,99), bone (n=910,38,48,54,59,63,69,75,78), and lung (n=730,38,63,71,78,79,89). All these studies reported shorter OS associated with the presence of metastasis at these specific sites compared to lack of it (eg, visceral vs non-visceral). Bone metastasis was also often used as the reference category when comparing the effect of other metastatic sites on survival, and was associated with improved prognosis compared to these other sites.10,44,54,69,75,78

Ten of 13 studies reported a significant association with PFS.26,29,30,32,33,36,60,63,76,84 Bone was the most assessed site (n=533,36,63,76,84), followed by liver (n=426,30,32,63), and visceral (n=430,60,63,76). As with OS, the presence of metastasis compared with absence in bone, liver, and visceral sites was associated with worse PFS; visceral sites reported worse PFS when compared with bone.10,78 Only one study reported poor tumor response associated with liver metastases.26

The definition of visceral sites varied across studies, most commonly defined as lung, liver, pericardial/pleural/peritoneal, and brain. Consistency in evidence and directionality of association was observed for OS and PFS. The overall effect size of association with survival was: moderate for liver, brain, and visceral sites; weak for lung; and ranged from weak to moderate for bone.

Time to recurrence or progression to advanced breast cancer was most often defined as the time between date of diagnosis of primary breast cancer, and date of diagnosis of first distant metastasis or recurrence. Disease-free interval (DFI), metastasis-free interval (MFI), and recurrence-free interval (RFI) are other terminology used to describe this. In Zhao et al,70 it was defined as the date from surgery to first recurrence. Eight studies did not report the definition.36,45,49,5254,66,71

The relationship between time to recurrence or progression to advanced breast cancer and OS (n=18) and PFS (n=5) was evaluated, with a significant association reported in 78% (n=14) and 80% (n=4) of studies, respectively.10,29,36,39,45,48,49,5254,60,66,70,71,91 In 13 studies, shorter time to recurrence or progression to advanced breast cancer was associated with worse survival relative to longer time, except in Jung et al,48 where the 15 years vs <1 year MFI was associated with worse OS (HR=1.30; 95% CI=1.021.65; P=0.032). The 2-year time interval was the most commonly studied cut-off point. Four studies showed a shorter time to recurrence or progression to advanced breast cancer (eg, <2 years) was associated with worse PFS.29,54,60,70 Consistency in evidence and directionality of association was observed for OS and PFS. The overall effect size of the association between time to recurrence or progression to advanced breast cancer and survival endpoints was moderate.

Given the patient population had advanced breast cancer, patients were likely to have received prior therapy (except those with de novo mBC) such as surgery, chemotherapy, radiation therapy, hormone therapy to treat early breast cancer. Type of prior therapy, line of prior therapy received in the metastatic setting, or clinical benefit to prior therapy were all grouped under the prior therapy category in this review.

Twenty-seven of 35 studies found a significant relationship between OS and prior therapy.10,25,28,30,33,34,38,44,49,52,54,55,57,58,60,66,71,72,76,79,80,8891,94 Prior therapy was either adjuvant or neoadjuvant chemotherapy or hormonal therapy in 19 studies that assessed OS.10,29,30,32,33,38,41,42,44,45,54,57,58,60,66,71,80,91,92 Lack of 1st-line hormonal therapy in patients with advanced breast cancer was also associated with worse survival compared with receiving hormonal therapy.80 Furthermore, the absence of hormonal maintenance therapy in the advanced setting was associated with worse OS in three studies.38,58,80 Two studies reported that adjuvant hormonal therapy use was associated with shorter survival compared with lack of use.33,54 Lobbezoo et al54 reported shorter survival was associated with receipt of initial chemotherapy compared with initial hormonal therapy in the metastatic setting.

Surgery was the prior therapy in ten studies that assessed OS.25,28,38,41,55,58,8890,94 Seven studies showed that receipt of surgery, compared with lack of surgery or best supportive care, resulted in significantly longer survival; five of these studies included de novo mBC patients28,55,89,90,94 and in the remaining two studies, surgery was conducted in early stage breast cancer.38,88

Prior radiotherapy was received in six studies that evaluated OS.38,41,66,79,82,89 First-line radiotherapy (yes vs no) was significantly associated with longer survival for mBC;38 however, the association was not uniform for 1st-line chemotherapy (multiagent vs none/singleagent); Li 2017 38 reported improved OS, while Xie et al33 reported worse OS.

Longer treatment durations in the advanced setting were associated with improved OS, while greater lines of treatment were associated with worse OS.66,76 Four studies demonstrated that the presence of clinical benefit or response to a specific treatment was associated with better OS.38,52,57,72

Fifteen studies assessed the association of PFS with line/type of prior therapy; 13 showed a significant relationship.23,27,29,30,32,33,38,47,54,58,60,70,76,84 Eleven of the 15 studies reported adjuvant or neoadjuvant chemotherapy or hormonal therapy,23,27,29,30,32,33,38,42,54,58,60 while four studies reported chemotherapy as prior treatment.47,70,76,84

Four studies compared multiple vs single lines of treatment and found that increasing treatment line in the metastatic setting correlated with worse prognosis.27,32,33,76 In two studies that included de novo patients, prognostic relevance was shown for surgery vs no surgery as prior therapy and found improved BCSS in patients undergoing breast-conserving surgery/mastectomy.55,90

There was substantial heterogeneity in reporting of type/class of therapy received. In general, patients receiving interventions (surgery/radiotherapy/systemic therapy), responding to treatments, or receiving fewer lines of treatment in the metastatic setting were likely to have better prognosis. Consistency in evidence and directionality of association was observed for OS, PFS, and BCSS. The effect size of the association between prior therapy attributes and survival endpoints was moderate.

Relationship between age and OS (n=37), PFS (n=7), and BCSS (n=3) was evaluated, with significant association reported in 46% (n=17), 29% (n=2), and 67% (n=2) of studies, respectively.10,28,30,33,36,38,43,48,55,57,58,62,69,78,84,85,9092 Among studies that found a significant association, increasing age was associated with worse OS, PFS, or BCSS. The time-point at which age data was collected in the study (whether age at diagnosis or at treatment initiation) was not reported in the majority of included studies. Among studies that did report, age at diagnosis was the most common. In three studies, increasing age was associated with worse OS.57,62,69 Age groups compared across studies varied widely (eg, >50 vs 50 years, >65, or 5064 vs 1849 years). Among the different age group comparisons, age 50 years was the most common cut-off point, reported in six studies.10,54,55,78,90,91 Less than 50% of studies found a significant association between age and OS as well as PFS, however, the directionality of association was consistent (ie, increasing age was associated with shorter survival). The effect size of the association between age and survival endpoints varied widely across studies, ranging from weak to strong.

Relationship between race and OS (n=13) and BCSS (n=3) was evaluated, with a significant association reported in 54% (n=7) and 100% (n=3) of studies, respectively.22,28,38,55,73,78,90 Poorer OS or BCSS was observed in blacks compared with whites. One study reported that better OS was observed for patients of other races vs whites (HR=0.59; 95% CI=0.440.78; P<0.001).38 One study evaluated but did not report a significant association between race and PFS.33 Consistency in evidence and directionality of association was observed between race and OS as well as BCSS. The effect size of the association between race and survival endpoints was weak.

Relationship between performance status and OS (n=14) and PFS (n=8) was evaluated, with a significant association reported in 79% (n=11) and 50% (n=4) of studies, respectively. ECOG scale was used in all but two studies; one study employed the World Health Organization (WHO) performance status scale,51 and one did not define the performance status scale.59 Comparison of different ECOG statuses varied across studies; most studies compared ECOG levels 2 vs 01, three studies compared 1 vs 0, while one study compared 3 vs 02.33,34,37,42,51,63,71,80 All studies found poor performance status or limitations in daily activity to be significantly associated with worse OS or PFS. Consistency in evidence and directionality of association was observed between performance status and OS. Less than 50% of studies found a significant association between PFS and performance status, however, the directionality of association was consistent. The effect size of the association between performance status and survival endpoints was moderate.

Table 2 summarizes the strength of evidence between prognostic factors and survival endpoints. Figure 2 shows the number of studies that reported better, worse, or no association between the prognostic factors and OS (Figure 2A) and PFS (Figure 2B).

Table 2 Strength of Evidence Assessment

Figure 2 Association between selected prognostic factors and OS (A), and PFS (B).

Abbreviations: BCSS, breast cancer-specific survival; CTC, circulating tumor cell; ECOG, Eastern Cooperative Oncology Group; OS, overall survival; PFS, progression-free survival; PR, progesterone receptor.

Associations between OS and PR status, tumor grade, CTC count, Ki67 level, de novo mBC, number and sites of metastases, time to recurrence or progression to advanced breast cancer, race, and prior therapy attributes were consistent (>50% of studies found a significant association). However, the evidence was limited (<50% of studies reported a significant association) for tumor size, histological type, lymph node involvement, and age. The direction of association was consistent for all the prognostic factors summarized in this study except for lymph node involvement. Based on effect size, strength of association with OS was moderate (HR=1.52.9) for PR status, tumor grade, Ki67 level, number and sites of metastases, time to recurrence or progression to advanced breast cancer, performance status, and prior therapy attributes, and weak (HR<1.5) for de novo metastatic breast cancer and race.

After applying the strongest evidence criteria, disease-related factors such as PR status, tumor grade, CTC count, Ki67 level, number and sites of metastases, and time to recurrence or progression to advanced breast cancer, performance status, prior therapy attributes, and race were found to have the strongest evidence of an association with OS.

Associations between PFS and tumor grade, CTC count, Ki67 level, number and sites of metastases, time to disease recurrence or progression to advanced breast cancer, and prior therapy attributes were consistent. However, the evidence was limited for PR status, lymph node involvement, histological type, performance status, age, and race; no data were reported for association between PFS and tumor size or marital status. The direction of association was consistent for all the prognostic factors, except for de novo metastatic breast cancer.

Since fewer studies assessed PFS than OS, evidence on prognostic factors related to PFS was limited. Thus, high CTC count, number and sites of metastases, and prior therapy attributes in the early or metastatic setting were the only four prognostic factors with the strongest evidence of an association with worse PFS. Similarly, there was limited information for the other endpoints.

There were many other variables assessed in the included studies. However, these were reported sparsely and we could not assess strength of evidence for them. They consisted of many genetic/biomarkers factors, for example, estrogen receptor gene (ESR1) mutation status,68 ligand binding domain (LBD) status,97 CA 153 level,51,70 alkaline phosphatase level,79 serum C-reactive protein level (CRP),79 lactic acid dehydrogenase (LDH) level,51,59,79 along with other demographic-related factors like marital status,55 income level,55 menopausal status,59 and education status.55 A high level summary can be found in Table 3.

The overall risk of bias was considered high for three studies, moderate for 22 studies, and low for the remaining 47 studies (Figure 3). Studies that failed to report exclusion criteria, definition of survival endpoints, or did not perform multivariate analysis to account for confounding were deemed high risk of bias.

Figure 3 Risk of bias assessment for each domain of QUIPS tool.

This comprehensive SLR was conducted to evaluate the strength and consistency of evidence of prognostic factors associated with survival in patients with HR+/HER2 advanced breast cancer. As commonly observed in oncology literature, OS was the most widely assessed survival endpoint, followed by PFS. The evidence was limited for tumor response (n=3) and BCSS (n=5). Hence, this review focused on prognostic factors associated with OS and PFS.

Higher CTC count, Ki67 level, number of metastases (multiple vs single), and sites of metastases (presence of liver metastases vs absence), prior therapy attributes, negative PR status, higher tumor grade, shorter time to recurrence or progression to advanced breast cancer, poor performance status and race (black vs white) were the prognostic factors with strongest evidence of association with OS and PFS. Previously published studies11,12,24,100,104 have also demonstrated the prognostic relationship between survival endpoints and disease-related factors such as PR status, CTC count, Ki67 level, number and sites of metastasis and treatment-related factors and performance status. Other studies in the literature103,105107 have also reported older age, black race, and unmarried status to be associated with shorter survival rates. Future cohort studies exclusively in HR+/HER2- advanced breast cancer will be beneficial to further validate the collective set of prognostic factors with the strongest evidence.

In the advance disease setting, breast cancer is incurable and the treatment goal is mainly palliative, improving quality-of-life and prolonging survival. Many factors are generally considered in developing treatment plans including patient-related factors like patient preferences, age, menopausal status, co-morbidities, performance status, socioeconomic status, psychological factors, treatment availabilities, and disease-related factors like DFI, previous therapies, tumor burden (number and sites), and any need for rapid disease control.108

This comprehensive review substantiates the importance of these factors in clinical decision-making for HR+/HER2 advanced breast cancer. The directionality of relationship between the prognostic factors and OS and PFS was largely consistent, except for lymph node involvement with OS and de novo metastatic breast cancer with PFS. Another published study109 also reported the divergent association between lymph node involvement and OS. A retrospective cohort study109 reported patients with N1 Stage IV BC had better OS than did those without lymph node metastasis (HR=0.902, 95% CI=0.8250.986, p-value=0.023). One potential explanation could be that the invasion of tumor cell into lymph nodes may have activated an antitumor immune response, which renders beneficial effect on patients with lymph node metastasis.110 Other studies106,111,112 have observed better OS in patients without lymph node metastasis compared to those with lymph node involvement. Similarly, the prognosis of de novo stage IV breast cancer was found to be better than those with recurrent tumors in several studies.4,113,114

Definitions of survival endpoints used across studies varied. The most common definition of OS was the time from diagnosis to death from any cause or last follow-up; many studies calculated the time interval from date of treatment initiation or patient selection. There was overlap in definitions of OS and BCSS. Gong et al55 defined BCSS as time from date of diagnosis to date of death attributed to breast cancer or date of last follow-up, while Yerushalmi et al93 defined BCSS as time from diagnosis of distant metastasis to death or censor date; two other studies did not define BCSS.22,90 It was observed that BCSS was not commonly assessed across included studies.

We observed heterogeneity in the comparison groups for certain prognostic factors for example, different age groups being compared (eg, >50 vs 50, >65, 5064 vs 1849); different cut-off points for Ki67 levels (10%, 14%, 25%, 30%); different prior therapies were compared (initial chemotherapy vs initial endocrine therapy, adjuvant endocrine therapy vs absence of prior therapy); site of metastasis (eg, presence vs absence of liver metastasis, visceral sites vs bone). Due to the differences in categorizations of prognostic factors as well as other factors such as differences in study design and patient population it was not possible to perform meta-analysis or derive a single hazard ratio estimate representing the relationship between the prognostic factors and survival endpoints. Despite inconsistencies in comparators groups, we observed an overall trend in directionality of association for some prognostic factors. For example, tumor size >5 cm diameter, CTC count 5/7.5 mL whole blood, time to recurrence or progression to advanced breast cancer of <2 years, and multiple vs single site of metastases were associated with worse survival.

This review focused on patients with HR+/HER2 advanced breast cancer; however, in 62% of 79 included studies, the proportion of patients with HR+/HER2 breast cancer ranged between 5079%. The results of such studies may not be reflective entirely of patients with HR+/HER2 advanced breast cancer. We observed a dearth of studies investigating the prognostic factors in exclusively patients with HR+/HER2 advanced breast cancer. For studies that included de novo metastatic patients, including in subgroups, baseline characteristics were captured in the metastatic setting. However, for the remaining studies, it was difficult to distinguish whether baseline characteristics were collected at initial diagnosis or when patients progressed to metastatic stage (as this information was not reported).

This review was subject to some limitations. An overall rating for risk of bias (low/moderate/high) was estimated for each study by taking into account the risk levels for the six domains of the QUIPS tool. The cut-off points chosen to derive the overall rating, though based on previously published SLRs, were essentially arbitrary.115117 Other limitations may be the exclusion of non-English studies, though English language studies from across the globe were included, and that studies published before 2010 and after 2018 were not included. Since the studies included in this SLR were published between 20102018, there were no studies that assessed the association between the newer targeted therapies such as CDK4/6 inhibitors, mTOR inhibitor, PI3K inhibitor, or kinase inhibitors, and survival endpoints. The conference abstracts included in this SLR contained limited relevant data and full-text publications related to these abstracts were not available. We found limited evidence on the prognostic value of genetic or tumor biomarkers in patients specifically with HR+/HER2 advanced BC. Some of these studies showed the relationship between tumor markers such as LDH,51,59,79 ALP,79 CEA,51,70 CA,51,70 to be associated with survival. This review did not distinguish the nature of the outcomes assessed (ie, primary or secondary) and therefore findings must be interpreted cautiously. Additionally, there was uncertainty around the power of subgroup analyses data reported in both observational studies and trials.

Strengths of this review include that this is the first SLR, to our knowledge, to comprehensively assess prognostic factors associated with survival in patients with HR+/HER2 advanced breast cancer. This review presents a complete overview of a large number of studies published recently with multivariate robust results that would help account for confounding of other key variables in understanding the association. This review was performed based on best practice guidelines, included supplementary searches of key conference proceedings and cross-referencing of other SLRs, and incorporated a double-blind study selection process, all of which lend to the robustness of this reviews methodology.

The strongest evidence for prognostic factors associated with worse OS included negative PR status, higher tumor grade, higher CTC count (5 vs <5), higher Ki67 levels (>14%), number of metastatic sites (multiple vs single), specific sites of metastases (presence of liver metastases vs absence), shorter time to recurrence or progression to advanced breast cancer, absence of prior therapy-related attributes (type of therapy, treatment line, response of prior therapy) in early or metastatic setting, poor performance status, and race (black vs white). The strongest evidence for prognostic factors associated with worse PFS included higher CTC count, number and sites of metastases, and prior therapy-related attributes in early or metastatic settings.

Apart from the commonly used markers recommended for routine use (eg, ER, PR, HER2), evaluation of the aforementioned factors shed light on the history and pathophysiology of the breast cancer in a patient, thereby providing a comprehensive clinical picture that may enable clinicians to enhance personalized treatment approaches and supportive care to improve patient outcomes. Identification of these prognostic factors will also guide future research in the HR+/HER2 advanced breast cancer setting.

We thank Michael Friedman for his editorial inputs.

ICON PLC. received funding from Eli Lilly and Company to conduct this review.

Keri Stenger, and Claudia Morato Guimares are employees and shareholders of Eli Lilly and Company. Gebra Cuyn Carter was an employee ofEli Lilly and Company when the review was being conducted. She is a shareholder of Eli Lilly and Company. Shereports personal fees from Exact Sciences.Maitreyee Mohanty, Shivaprasad Singuru, Vanita Tongbram are employees of ICON PLC.Pradeep Basa and Sheena Singh were employees of ICON PLC. when the review was being conducted.Dr. Kuemmel reports personal fees from Eli Lilly and Company, Roche, Genomic Health, Novartis, Amgen, Celgene, Daiichi Sankyo, Sonoscope, AstraZeneca, Somatex, MSD, Pfizer, Puma Biotechnology, PFM medical, non-financial support from Roche, Daiichi Sankyo outside the submitted work. Dr. Guarneri reports personal fees from Eli Lilly and Company, Novartis, Roche, MSD, outside the submitted work. Dr Tolaney reports grants and personal fees Eli Lilly and Company, AstraZeneca, Merck, Nektar, Novartis, Pfizer, Genentech/Roche, Immunomedics, Exelixis, Bristol-Myers Squibb, Eisai, Nanostring, Puma, Cyclacel, sanofi, Celldex, Odonate, Seattle Genetics, Silverback Therapeutics, G1 Therapeutics, AbbVie, Anthenex, OncoPep, Kyowa Kirin Pharmaceuticals, Daiichi-Sankyo, Mersana Therapeutics, Certara,CytomX, Samsung Bioepsis Inc., Gilead, outside the submitted work.

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Prognostic factors in patients with HR+/HER2 breast cancer | CMAR - Dove Medical Press

Hartford Services – FXDFR DB REDEEM 15/04/2042 USD 25 (HGH) gains 0.15% in Active Trading on August 6 – Equities.com

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